Imagine a study that has tested the effects of spider toxins on action potential propagation in axons of crickets. When the toxin is applied to the axon, the propagation of action potentials is modified so that the amplitude of action potentials is reduced by 80% from 90 mV to 18 mV. Voltage-gated sodium and potassium channels are not affected by the toxin. The toxin does not activate or inhibit either one of the two channels that generate the action potential. It cannot be ruled out that an unknown ion channel is targeted by the toxin. What unknown mechanism could cause the 80% reduction in the action potential amplitude in the axon? Explain