Reset When a blood vessel is damaged, the fibers within the connective tissue beneath the endothelial cells in the vessel wall become exposed. Platelets adhere to these fibers with the assistance of a plasma protein called . As the platelets start to stick to the vessel wall, their morphology changes dramatically. They develop that further adhere them to the blood vessel wall. As more and more platelets aggregate to the site, a(n) develops to close off the injury. This is a measure to block the flow of blood to an area where a vessel wall is damaged. Platelets undergo this morphologic change and become activated. Their cytoplasm releasing chemicals to assist with hemostasis.

you are certainly talking about this picture:

a: is the label of the white square, it is showing the  replication fork, formed by helicase (d) ==> so it is the site of dna separation and replication 

b: is a new dna brand that is following directly the replication fork, so it is continuous ==> built continuously from 5' to 3

c: dna polymerase cannot replicate dna on its own, it needs the of their proteins, and one of them plays the role of a clamp which fixes  the dna with the dna template and increases  the processivity of the enzyme. ==>   stops polymerase from falling off the  template.

d: is the enzyme responsible for forming the replication fork (helicase) ==> unwinds and separates dna into two strands

e: is the delayed strand, it is built  in the opposite sense of the helicase, so dna will be built in fragments ==> built  fragments from 5' to 3 joins

and finally f: is the enzyme that builds  the new dna strand (dna polymerase) ==>   adds nucleotides to make a new dna helix.

credit: gradman555p98x9p

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