Inherited defects in components of purine catabolism and salvage are associated with various conditions and syndromes. The enzyme hypoxanthine-guanine (HGPRT) is a key enzyme in the purine salvage pathway. It is responsible for reforming IMP and GMP from hypoxanthine and guanine, respectively. In this manner purine bases are salvaged back into the purine nucleotide pool. Genetic defects that lead to the loss of HGPRT activity are the primary cause for which of the following conditions? a. Gout
b. Lesch-Nyhan syndrome
c. Orotic aciduria
d. Severe combined immunodeficiency syndrome
e. Tay-Sachs disease

Ireally dont know sorry

Atp is is our little "pocket" of energy. of course, if you have too little, then it means you have too little energy, and you'll pass out. the way to not have enough, is to have no energy sources (glucose, fats, proteins). you can't have too many atp molecules. cycles like the krebs cycle produce it, but if you have too much of any substrate (or too little for that matter), the cycle stops and atp is no longer made (it stops until substrate levels are such that the process can go on). in any case, this is unlikely since atp is constantly hydrolyzed and remade, even while you're asleep. you won't have too much. if you have zero, you are dead or dying.

The answer is c) organisms that are sensitive to changes in temperature would die.

A. convection currents in the mantle.is the correct answer