Sometimes after an intense exercise routine, an individual will have sore muscles 1 to 2 days following the exercise; this is called delayed onset muscle soreness. Although not completely understood, it is believed that one cause of this is damage that occurs to the Z disks and contractile filaments in the muscles. When the body attempts to repair this damage, an inflammatory response occurs that causes soreness a couple of days after the exercise has taken place. Which of these situations would likely lead to less delayed onset muscle soreness in an individual? a. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be stronger
b. a mutation that would reduce the amount of lactic acid buildup in muscle tissue
c. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be weaker
d. a mutation that weakens the calcium binding ability of troponin

The correct answer is option A. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be stronger

Explanation:

As delayed onset muscle soreness is thought to be led by the inflammatory response of the body in order to repair damage to the Z disks and contractile filaments in muscles, due to the intense exercise.

Mutation in the Z-disk protein would strengthen the connection between Z disks and contractile filaments as it is logical that this mutation could withstand more pressure or intense exercise before damage occurs to these tissues.

the oxygenated blood then leaves the lungs through pulmonary veins, which return it to the left heart, completing the pulmonary cycle. this blood then enters the left atrium, which pumps it through the mitral valve into the left ventricle. from the left ventricle, the blood passes through the aortic valve to the aorta.

deoxygenated blood leaves through the right ventricle through the pulmonary artery. from the right atrium, the blood is pumped through the tricuspid valve (or right atrioventricular valve), into the right ventricle.