Aclient with long qt syndrome has the gene that increases potassium channels. how will this affect membrane potentials?

Delayed return to resting potential.

Explanation:

LQTS (Long QT syndrome) may be defined as the medical condition in which the heart's electrical conductivity gets dis balance. This might occur due to the exercise in case of arrhythmias and quite uncontrollable.

The resting potential of the call is around -70mV. This is maintained by the presence of the potassium ions. In case of LQTS, there is delay in returning to the resting membrane potential of the cell. The cell remains for long in the depolarization phase and may affect the heart's conductivity.

Thus, the answer is delayed return to resting potential.

the oxygenated blood then leaves the lungs through pulmonary veins, which return it to the left heart, completing the pulmonary cycle. this blood then enters the left atrium, which pumps it through the mitral valve into the left ventricle. from the left ventricle, the blood passes through the aortic valve to the aorta.

deoxygenated blood leaves through the right ventricle through the pulmonary artery. from the right atrium, the blood is pumped through the tricuspid valve (or right atrioventricular valve), into the right ventricle.

yes it is weird but true