Agenetic form of "night blindness" (i. e. poor vision in dim light) is caused by mutations in genes encoding rhodopsin kinase (rk) and arrestin. the mutations reduce the dynamic range of light perception, and the affected individuals have a prolonged light response and adapt very slowly to low light (when entering a dark theater room, for example). which of the following mutations do you expect to be the cause? a. loss-of-function rk mutations and loss-of-function arrestin mutations b. loss-of-function rk mutations and gain-of-function arrestin mutations c. gain-of-function rk mutations and loss-of-function arrestin mutations d. gain-of-function rk mutations and gain-of-function arrestin mutations
Answers: 3
Biology, 21.06.2019 15:00, alexisolsennn4680
In familial hypercholesterolemia individuals homozygous for the allele causing the disorder completely lack receptors on liver cells that take up cholesterol from the blood stream. heterozygous have one-half the number of receptors while individuals homozygous for the normal allele are phenotypically normal. this is an example of a. epistasis b. complete dominance c. incomplete dominance d. codominance
Answers: 2
Biology, 22.06.2019 02:00, sanakhalid8881
The concept of keystone species is controversial among ecologists because most organisms are highly interdependent. if each of the trophic levels is dependant on all others how can we say one is most important
Answers: 3
Agenetic form of "night blindness" (i. e. poor vision in dim light) is caused by mutations in genes...
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